text_1: Increased microtubule acetylation repairs LRRK2 Roc-COR domain mutation induced locomotor deficits.
text_2: Here we find that LRRK2 containing pathogenic Roc-COR domain mutations (R1441C, Y1699C) preferentially associates with deacetylated microtubules, and inhibits axonal transport in primary neurons and in Drosophila, causing locomotor deficits in vivo.